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Clinical Examples

Pharmacogenomics in Practice

Explore real-world scenarios where pharmacogenomics transforms treatment outcomes.

Codeine + CYP2D6
CYP2D6

Codeine

Clinical Risk

Poor Metabolizers: No pain relief (codeine inactive); Ultra-Rapid Metabolizers: Overdose risk (morphine toxicity)

Recommendation

Test CYP2D6 before prescribing. Poor metabolizers: avoid. Ultra-rapid metabolizers: avoid or max 30mg dose.

The Science

Codeine is a prodrug converted to morphine by CYP2D6. Your metabolizer status determines efficacy and safety.

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Clopidogrel + CYP2C19
CYP2C19

Clopidogrel

Clinical Risk

Poor Metabolizers: Reduced platelet inhibition; stent thrombosis; heart attack

Recommendation

Poor metabolizers: avoid clopidogrel. Use prasugrel or ticagrelor instead (active drugs, not prodrugs).

The Science

Clopidogrel is a prodrug requiring CYP2C19 activation. Poor metabolizers receive minimal benefit and face higher cardiovascular risk.

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Warfarin + CYP2C9 & VKORC1
CYP2C9 & VKORC1

Warfarin

Clinical Risk

Patients with variants in CYP2C9 (*2, *3) or VKORC1 (-1639G>A): Excessive INR elevation, bleeding

Recommendation

FDA recommends PGx-guided dosing. Variants = lower starting dose, frequent INR monitoring.

The Science

Warfarin response is highly polygenic. Testing CYP2C9 and VKORC1 improves dosing accuracy and reduces bleeding events.

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Simvastatin + SLCO1B1
SLCO1B1

Simvastatin

Clinical Risk

Variants (521T>C, 388A>G): Increased statin levels, muscle pain, rhabdomyolysis

Recommendation

Carriers: avoid simvastatin >20mg. Use pravastatin or rosuvastatin instead (no SLCO1B1 transport).

The Science

SLCO1B1 transports simvastatin into liver cells. Variants reduce clearance, increasing myopathy risk.

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SSRIs + CYP2D6 or CYP2C19
CYP2D6 or CYP2C19

Paroxetine, Fluoxetine, Escitalopram

Clinical Risk

Poor Metabolizers: Excessive plasma levels, serotonin syndrome, QT prolongation, seizures

Recommendation

Poor metabolizers: lower starting dose; frequent symptom monitoring; consider alternative SSRI or tricyclic.

The Science

SSRIs are metabolized by CYP2D6 or CYP2C19. Poor metabolizers experience side effects at standard doses.

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Azathioprine + TPMT
TPMT

Azathioprine

Clinical Risk

Poor Metabolizers: Severe bone marrow suppression, infections, aplastic anemia; Heterozygotes: moderate toxicity

Recommendation

MANDATORY testing before use. Poor metabolizers: avoid or use 10% of standard dose.

The Science

Azathioprine is converted to thioguanine nucleotides. TPMT poor metabolizers cannot clear these toxic metabolites.

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Carbamazepine + HLA-B*15:02
HLA-B*15:02

Carbamazepine

Clinical Risk

Stevens-Johnson Syndrome (SJS) or Toxic Epidermal Necrolysis (TEN); mortality up to 30%

Recommendation

Screen before use, especially in East/Southeast Asian populations. HLA-B*15:02 carriers: avoid carbamazepine; use alternative anticonvulsant.

The Science

HLA-B*15:02 is a strong predictor of severe cutaneous reactions to carbamazepine. FDA recommends screening.

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Abacavir + HLA-B*5701
HLA-B*5701

Abacavir

Clinical Risk

HLA-B*5701 carriers: Hypersensitivity reaction (fever, rash, GI, respiratory symptoms) within 1-3 weeks

Recommendation

Mandatory screening before initiation. HLA-B*5701 positive: contraindicated; use alternative antiretroviral.

The Science

Abacavir triggers immune hypersensitivity in 5-8% of patients. Genetic screening prevents life-threatening reactions.

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For Healthcare Professionals: These scenarios are educational summaries. Always consult full CPIC guidelines, FDA labels, and clinical evidence when making treatment decisions. Each patient's care must be individualized.